Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome

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ORPHA:457205G60.8
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Overview

Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome is an extremely rare genetic condition that affects the nervous system starting in infancy. This disease damages the peripheral nerves (the nerves that carry signals between the brain, spinal cord, and the rest of the body), leading to both motor problems (difficulty moving muscles) and sensory problems (difficulty feeling touch, temperature, or pain). It also causes optic atrophy, which means the nerve connecting the eye to the brain gradually deteriorates, leading to vision loss. Over time, the condition causes progressive neurodegeneration, meaning the brain and nervous system continue to lose function. Babies with this condition typically show early signs such as weak muscle tone (floppiness), difficulty feeding, and delayed motor milestones like sitting or walking. As the disease progresses, children may experience worsening muscle weakness, loss of sensation in the hands and feet, vision problems, and developmental regression — meaning they may lose skills they had previously gained. Some children may also develop breathing difficulties and intellectual disability. This syndrome is caused by mutations in the MFN2 gene, which plays a critical role in how mitochondria (the energy-producing structures inside cells) function and fuse together. Because there is currently no cure, treatment focuses on managing symptoms and supporting quality of life. This may include physical therapy, occupational therapy, assistive devices for mobility, and regular monitoring by a team of specialists. The condition is classified under ICD-10 code G60.8 (other hereditary and idiopathic neuropathies).

Also known as:

Key symptoms:

Muscle weakness starting in infancyLow muscle tone (floppiness)Loss of sensation in hands and feetVision loss due to optic nerve damageDelayed motor milestonesLoss of previously learned skills (developmental regression)Difficulty walking or inability to walkMuscle wasting (thin, weak muscles)Difficulty feedingBreathing problemsReduced or absent reflexesIntellectual disabilityPoor balance and coordinationFoot deformities such as high arches or curled toes

Clinical phenotype terms (26)— hover any for plain English
Sensory axonal neuropathyHP:0003390Motor axonal neuropathyHP:0007002Horizontal supranuclear gaze palsyHP:0007817Foot joint contractureHP:0008366
Inheritance

Autosomal recessive

Passed on when both parents carry the same gene change; often skips generations

Age of Onset

Infantile

Begins in infancy, roughly 1 month to 2 years old

Orphanet ↗NORD ↗

Treatments

No FDA-approved treatments are currently listed for Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome.

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No actively recruiting trials found for Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome at this time.

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No specialists are currently listed for Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome.

View NORD Rare Disease Centers ↗Undiagnosed Disease Network ↗

Treatment Centers

8 centers
🏥 NORD

Baylor College of Medicine Rare Disease Center

Baylor College of Medicine

📍 Houston, TX

🏥 NORD

Stanford Medicine Rare Disease Center

Stanford Medicine

📍 Stanford, CA

🔬 UDN

NIH Clinical Center Undiagnosed Diseases Program

National Institutes of Health

📍 Bethesda, MD

🔬 UDN

UCLA UDN Clinical Site

UCLA Health

📍 Los Angeles, CA

🔬 UDN

Baylor College of Medicine UDN Clinical Site

Baylor College of Medicine

📍 Houston, TX

🔬 UDN

Harvard/MGH UDN Clinical Site

Massachusetts General Hospital

📍 Boston, MA

🏥 NORD

Mayo Clinic Center for Individualized Medicine

Mayo Clinic

📍 Rochester, MN

👤 Mayo Clinic Center for Individualized Medicine

🏥 NORD

UCLA Rare Disease Day Program

UCLA Health

📍 Los Angeles, CA

Travel Grants

No travel grants are currently matched to Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome.

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Community

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Questions for your doctor

Bring these to your next appointment

  • Q1.What is the expected rate of progression for my child's condition?,What therapies should we start right away to help maintain function?,How often should my child have eye exams and neurological evaluations?,Are there any clinical trials or experimental treatments we should consider?,What signs of breathing problems should I watch for at home?,Should other family members be tested for carrier status?,What support services and resources are available for our family?

Common questions about Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome

What is Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome?

Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome is an extremely rare genetic condition that affects the nervous system starting in infancy. This disease damages the peripheral nerves (the nerves that carry signals between the brain, spinal cord, and the rest of the body), leading to both motor problems (difficulty moving muscles) and sensory problems (difficulty feeling touch, temperature, or pain). It also causes optic atrophy, which means the nerve connecting the eye to the brain gradually deteriorates, leading to vision loss. Over time, the condi

How is Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome inherited?

Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome follows a autosomal recessive inheritance pattern. Genetic counseling can help families understand recurrence risk and testing options.

At what age does Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome typically begin?

Typical onset of Infantile-onset axonal motor and sensory neuropathy-optic atrophy-neurodegenerative syndrome is infantile. Age of onset can vary across affected individuals.