Hyperinsulinism-hyperammonemia syndrome

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ORPHA:35878OMIM:606762E72.8
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1Specialists8Treatment centers

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Overview

Hyperinsulinism-hyperammonemia syndrome (HI/HA syndrome) is a rare inherited metabolic disorder that causes two main problems at the same time: too much insulin in the blood and too much ammonia in the blood. Insulin is the hormone that lowers blood sugar, and when there is too much of it, blood sugar can drop dangerously low — a condition called hypoglycemia. Ammonia is a waste product that the body normally clears away, and when it builds up, it can affect the brain. This condition is also sometimes called glutamate dehydrogenase hyperinsulinism or GLUD1 hyperinsulinism. The syndrome is caused by changes (mutations) in a gene called GLUD1, which controls an enzyme involved in processing certain amino acids, especially after eating protein-rich foods. Because of this, meals high in protein can trigger episodes of low blood sugar. These episodes can cause shakiness, sweating, confusion, and in severe cases, seizures or loss of consciousness. With proper treatment and careful dietary management, many people with HI/HA syndrome can live relatively normal lives. However, some children may develop learning difficulties or epilepsy, especially if low blood sugar episodes are not caught and treated quickly. Early diagnosis and ongoing medical care are very important for the best possible outcome.

Also known as:

Key symptoms:

Low blood sugar (hypoglycemia), especially after eating proteinShakiness or tremblingSweating without obvious causeConfusion or difficulty thinking clearlySeizures or convulsionsElevated ammonia levels in the bloodIrritability or unusual fussiness in infantsPale or clammy skinDifficulty concentrating or learning problemsFatigue or low energyFainting or loss of consciousness in severe episodes

Clinical phenotype terms (12)— hover any for plain English
Asymptomatic hyperammonemiaHP:0008162Reactive hypoglycemiaHP:0012051Generalized non-motor (absence) seizureHP:0002121Fasting hyperinsulinemiaHP:0008283EEG with generalized epileptiform dischargesHP:0011198Increased urine alpha-ketoglutarate concentrationHP:0012402
Inheritance

Autosomal dominant

Passed on from just one parent; each child has about a 50% chance of inheriting it

Age of Onset

Infantile

Begins in infancy, roughly 1 month to 2 years old

Orphanet ↗OMIM ↗NORD ↗

Treatments

No FDA-approved treatments are currently listed for Hyperinsulinism-hyperammonemia syndrome.

View clinical trials →

No actively recruiting trials found for Hyperinsulinism-hyperammonemia syndrome at this time.

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Specialists

1 foundView all specialists →
EM
Elizabeth Rosenfeld, MD
NEW YORK, NY
Specialist
PI on 1 active trial

Treatment Centers

8 centers
🏥 NORD

Baylor College of Medicine Rare Disease Center

Baylor College of Medicine

📍 Houston, TX

🏥 NORD

Stanford Medicine Rare Disease Center

Stanford Medicine

📍 Stanford, CA

🔬 UDN

NIH Clinical Center Undiagnosed Diseases Program

National Institutes of Health

📍 Bethesda, MD

🔬 UDN

UCLA UDN Clinical Site

UCLA Health

📍 Los Angeles, CA

🔬 UDN

Baylor College of Medicine UDN Clinical Site

Baylor College of Medicine

📍 Houston, TX

🔬 UDN

Harvard/MGH UDN Clinical Site

Massachusetts General Hospital

📍 Boston, MA

🏥 NORD

Mayo Clinic Center for Individualized Medicine

Mayo Clinic

📍 Rochester, MN

👤 Mayo Clinic Center for Individualized Medicine

🏥 NORD

UCLA Rare Disease Day Program

UCLA Health

📍 Los Angeles, CA

Travel Grants

No travel grants are currently matched to Hyperinsulinism-hyperammonemia syndrome.

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Community

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Caregiver Resources

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Questions for your doctor

Bring these to your next appointment

  • Q1.How often should we check blood sugar at home, and what levels should prompt us to act?,What is the right dose of diazoxide for my child, and what side effects should I watch for?,How much protein is safe to eat at one meal, and are there specific foods we should avoid?,Does my child need neurological or developmental testing because of the high ammonia levels?,What should we do in an emergency if my child loses consciousness or has a seizure?,Should other family members be tested for this condition?,Are there any clinical trials or new treatments we should know about?

Common questions about Hyperinsulinism-hyperammonemia syndrome

What is Hyperinsulinism-hyperammonemia syndrome?

Hyperinsulinism-hyperammonemia syndrome (HI/HA syndrome) is a rare inherited metabolic disorder that causes two main problems at the same time: too much insulin in the blood and too much ammonia in the blood. Insulin is the hormone that lowers blood sugar, and when there is too much of it, blood sugar can drop dangerously low — a condition called hypoglycemia. Ammonia is a waste product that the body normally clears away, and when it builds up, it can affect the brain. This condition is also sometimes called glutamate dehydrogenase hyperinsulinism or GLUD1 hyperinsulinism. The syndrome is cau

How is Hyperinsulinism-hyperammonemia syndrome inherited?

Hyperinsulinism-hyperammonemia syndrome follows a autosomal dominant inheritance pattern. Genetic counseling can help families understand recurrence risk and testing options.

At what age does Hyperinsulinism-hyperammonemia syndrome typically begin?

Typical onset of Hyperinsulinism-hyperammonemia syndrome is infantile. Age of onset can vary across affected individuals.

Which specialists treat Hyperinsulinism-hyperammonemia syndrome?

1 specialists and care centers treating Hyperinsulinism-hyperammonemia syndrome are listed on UniteRare, sourced from ClinicalTrials.gov principal investigators, published research, and the NPPES NPI registry.