Familial progressive hyper- and hypopigmentation

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ORPHA:280628OMIM:145250L81.8
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Overview

Familial progressive hyper- and hypopigmentation (FPHH) is a rare inherited skin condition that causes patches of both darker (hyperpigmented) and lighter (hypopigmented) skin to develop over time. The condition typically begins in infancy or early childhood and gradually progresses, meaning the patches of uneven skin color tend to spread and become more noticeable as a person grows older. The pigmentation changes can appear anywhere on the body, including the trunk, arms, legs, and face. FPHH is caused by changes (mutations) in genes that help control how melanin — the pigment that gives skin its color — is distributed in the skin. The condition is generally not associated with problems in other organs, meaning it primarily affects the skin's appearance rather than overall health. However, the visible skin changes can have a significant emotional and psychological impact, especially during childhood and adolescence. There is currently no cure for FPHH. Treatment is mainly supportive and cosmetic, focusing on sun protection and, in some cases, cosmetic camouflage products to even out skin tone. Dermatologists and geneticists are the main specialists involved in managing this condition. Because it is so rare, awareness among healthcare providers may be limited, and diagnosis can sometimes be delayed.

Also known as:

Key symptoms:

Patches of darker skin (hyperpigmentation)Patches of lighter skin (hypopigmentation)Uneven or mottled skin colorGradual worsening of skin color changes over timeSkin changes that can appear anywhere on the bodyFreckle-like spots on the skinCafé-au-lait spots (light brown flat birthmarks)Skin changes present from early childhoodNo associated pain or itching in most cases

Inheritance

Autosomal dominant

Passed on from just one parent; each child has about a 50% chance of inheriting it

Age of Onset

Infantile

Begins in infancy, roughly 1 month to 2 years old

Orphanet ↗OMIM ↗NORD ↗

Treatments

1 available

Secreflo

Synthetic porcine secretin· ChiRhoClin, Inc.
For use in secretin stimulation testing for: Stimulation of pancreatic secretions to facilitate the identification of the ampulla of Vater and accessory papilla during endoscopic retrograde cholangio-

For use in secretin stimulation testing for: Stimulation of pancreatic secretions to facilitate the identification of the ampulla of Vater and accessory papilla during endoscopic retrograde cholangio-pancreatography (ERCP)

No actively recruiting trials found for Familial progressive hyper- and hypopigmentation at this time.

New trials open frequently. Follow this disease to get notified.

Search ClinicalTrials.gov ↗Join the Familial progressive hyper- and hypopigmentation community →

No specialists are currently listed for Familial progressive hyper- and hypopigmentation.

View NORD Rare Disease Centers ↗Undiagnosed Disease Network ↗

Treatment Centers

8 centers
🏥 NORD

Baylor College of Medicine Rare Disease Center

Baylor College of Medicine

📍 Houston, TX

🏥 NORD

Stanford Medicine Rare Disease Center

Stanford Medicine

📍 Stanford, CA

🔬 UDN

NIH Clinical Center Undiagnosed Diseases Program

National Institutes of Health

📍 Bethesda, MD

🔬 UDN

UCLA UDN Clinical Site

UCLA Health

📍 Los Angeles, CA

🔬 UDN

Baylor College of Medicine UDN Clinical Site

Baylor College of Medicine

📍 Houston, TX

🔬 UDN

Harvard/MGH UDN Clinical Site

Massachusetts General Hospital

📍 Boston, MA

🏥 NORD

Mayo Clinic Center for Individualized Medicine

Mayo Clinic

📍 Rochester, MN

👤 Mayo Clinic Center for Individualized Medicine

🏥 NORD

UCLA Rare Disease Day Program

UCLA Health

📍 Los Angeles, CA

Travel Grants

No travel grants are currently matched to Familial progressive hyper- and hypopigmentation.

Search all travel grants →NORD Financial Assistance ↗

Community

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Latest news about Familial progressive hyper- and hypopigmentation

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Caregiver Resources

NORD Caregiver Resources

Support, advocacy, and financial assistance for caregivers of rare disease patients.

Mental Health Support

Rare disease caregiving can be isolating. Connect with counseling and peer support.

Family & Caregiver Grants

Financial assistance programs specifically for caregivers of rare disease patients.

Social Security Disability

Learn how rare disease patients may qualify for SSDI/SSI benefits.

Questions for your doctor

Bring these to your next appointment

  • Q1.Is genetic testing recommended for my child, and what gene should be tested?,How is this condition likely to progress over time?,Are there any treatments that can slow down or reduce the pigmentation changes?,Should other family members be evaluated or tested?,What sun protection measures do you recommend?,Can you refer us to a counselor experienced with visible skin differences?,Are there any clinical trials or research studies we could participate in?

Common questions about Familial progressive hyper- and hypopigmentation

What is Familial progressive hyper- and hypopigmentation?

Familial progressive hyper- and hypopigmentation (FPHH) is a rare inherited skin condition that causes patches of both darker (hyperpigmented) and lighter (hypopigmented) skin to develop over time. The condition typically begins in infancy or early childhood and gradually progresses, meaning the patches of uneven skin color tend to spread and become more noticeable as a person grows older. The pigmentation changes can appear anywhere on the body, including the trunk, arms, legs, and face. FPHH is caused by changes (mutations) in genes that help control how melanin — the pigment that gives ski

How is Familial progressive hyper- and hypopigmentation inherited?

Familial progressive hyper- and hypopigmentation follows a autosomal dominant inheritance pattern. Genetic counseling can help families understand recurrence risk and testing options.

At what age does Familial progressive hyper- and hypopigmentation typically begin?

Typical onset of Familial progressive hyper- and hypopigmentation is infantile. Age of onset can vary across affected individuals.