3-methylglutaconic aciduria type 9

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ORPHA:505216OMIM:617698E71.1
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8Treatment centers

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Overview

3-methylglutaconic aciduria type 9 (also known as MGCA9 or 3-MGA type 9) is a very rare inherited metabolic disorder caused by changes in the CLPB gene. This gene provides instructions for making a protein that helps cells manage stress and keep other proteins working properly, especially in the mitochondria — the parts of cells that produce energy. When this protein does not work correctly, harmful substances build up in the body, and cells — particularly in the brain and blood — cannot function normally. The condition mainly affects the nervous system and the blood. Children with MGCA9 often have serious neurological problems, including movement difficulties, intellectual disability, and seizures. Many also develop a dangerous drop in white blood cell counts (neutropenia), which makes fighting infections very difficult. Other features can include cataracts (clouding of the eye lens) and problems with muscle tone. Symptoms usually appear in infancy or early childhood and can be life-threatening. There is currently no cure for 3-methylglutaconic aciduria type 9. Treatment focuses on managing symptoms, preventing infections, and supporting development. Care typically involves a team of specialists including metabolic geneticists, neurologists, and blood specialists. Early diagnosis through genetic testing is important so that families can access the right support and monitoring as quickly as possible.

Also known as:

Key symptoms:

Low white blood cell count (neutropenia), making infections more likelyIntellectual disability or developmental delayMovement problems such as poor coordination or unsteady walking (ataxia)Muscle stiffness or abnormal muscle toneSeizuresClouding of the eye lens (cataracts)Abnormal levels of 3-methylglutaconic acid in the urineFeeding difficulties in infancyDelayed speech and language developmentRecurrent or severe infections due to low immunity

Clinical phenotype terms (25)— hover any for plain English
Slender buildHP:00015333-Methylglutaconic aciduriaHP:0003535Diffuse white matter abnormalitiesHP:0007204Decreased activity of mitochondrial ATP synthase complexHP:0011925
Inheritance

Autosomal recessive

Passed on when both parents carry the same gene change; often skips generations

Age of Onset

Infantile

Begins in infancy, roughly 1 month to 2 years old

Orphanet ↗OMIM ↗NORD ↗

Treatments

No FDA-approved treatments are currently listed for 3-methylglutaconic aciduria type 9.

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No actively recruiting trials found for 3-methylglutaconic aciduria type 9 at this time.

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No specialists are currently listed for 3-methylglutaconic aciduria type 9.

View NORD Rare Disease Centers ↗Undiagnosed Disease Network ↗

Treatment Centers

8 centers
🏥 NORD

Baylor College of Medicine Rare Disease Center

Baylor College of Medicine

📍 Houston, TX

🏥 NORD

Stanford Medicine Rare Disease Center

Stanford Medicine

📍 Stanford, CA

🔬 UDN

NIH Clinical Center Undiagnosed Diseases Program

National Institutes of Health

📍 Bethesda, MD

🔬 UDN

UCLA UDN Clinical Site

UCLA Health

📍 Los Angeles, CA

🔬 UDN

Baylor College of Medicine UDN Clinical Site

Baylor College of Medicine

📍 Houston, TX

🔬 UDN

Harvard/MGH UDN Clinical Site

Massachusetts General Hospital

📍 Boston, MA

🏥 NORD

Mayo Clinic Center for Individualized Medicine

Mayo Clinic

📍 Rochester, MN

👤 Mayo Clinic Center for Individualized Medicine

🏥 NORD

UCLA Rare Disease Day Program

UCLA Health

📍 Los Angeles, CA

Travel Grants

No travel grants are currently matched to 3-methylglutaconic aciduria type 9.

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Community

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Caregiver Resources

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Questions for your doctor

Bring these to your next appointment

  • Q1.How often should my child's white blood cell count be checked, and what level should trigger emergency care?,Is my child a candidate for G-CSF treatment or bone marrow transplantation, and what are the risks and benefits?,What developmental therapies do you recommend, and how do we access them?,What is the emergency plan if my child develops a fever or signs of serious infection?,Should other family members be tested to see if they are carriers of the CLPB gene change?,Are there any research studies or clinical trials we should know about?,What signs of disease progression should we watch for, and when should we contact you urgently?

Common questions about 3-methylglutaconic aciduria type 9

What is 3-methylglutaconic aciduria type 9?

3-methylglutaconic aciduria type 9 (also known as MGCA9 or 3-MGA type 9) is a very rare inherited metabolic disorder caused by changes in the CLPB gene. This gene provides instructions for making a protein that helps cells manage stress and keep other proteins working properly, especially in the mitochondria — the parts of cells that produce energy. When this protein does not work correctly, harmful substances build up in the body, and cells — particularly in the brain and blood — cannot function normally. The condition mainly affects the nervous system and the blood. Children with MGCA9 ofte

How is 3-methylglutaconic aciduria type 9 inherited?

3-methylglutaconic aciduria type 9 follows a autosomal recessive inheritance pattern. Genetic counseling can help families understand recurrence risk and testing options.

At what age does 3-methylglutaconic aciduria type 9 typically begin?

Typical onset of 3-methylglutaconic aciduria type 9 is infantile. Age of onset can vary across affected individuals.